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In ovaries of the Dicer-2 mutant, we detected only 26 31 nt long piRNAs, whereas 19 25 nt long siRNAs were also detected in the wild type ovaries.
In the mutant, we detected a complex pattern of deletion/rearrangements at the pro41 locus.
In transient transfections conducted with the D-box1 mutant, we detected only L-Snord115 RNA in northern blots (fig. 2 A, B lane 5; fig. 2 D).
In this mutant, we detected only one or two vCrz-sib neurons (Fig. 10H), which is in stark contrast to the results of the dark CD4/82 larvae from dark CD4/CD4 mother (Fig. 10G).
Among the genes that are up-regulated in the zur mutant, we detected five C. glutamicum transcription units that are preceded by candidate Zur-binding sites: cg2911- cg2912-cg2913, cg0040-cg0041-cg0042/ cg0043 and cg0794/cg0795 (Table 1).
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In the double-headed MORN1 mutants we detected the apical Centrin2 structures on both ends of the parasite (Fig. 5L).
However, when investigating the larval development of Drosophila C3G mutants, we detected substantial disorganization of the larval muscle architecture, as well as defects in integrin localization at muscle attachment sites.
In the single knockout mutants we detected a ∼50% loss of hybridization signal from DNA fragments encoding the respective genes, but there was no detectable hybridization in the knockout cells where both alleles had been removed (Figure 3B).
In the fiber sap of short fiber mutants we detected significantly lower osmotic pressure than in WT.
In double mutants, we detected again a prominent reduction in spine density compared to dendrites of APLP2-KO and WT neurons.
Of the 2847 prototroph mutants, we detected 75%and65%5% with ≥1 or ≥10 sequence reads, respectively, at timepoint zero for each of the replicates.
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