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We used a trans-complementation-based system to demonstrate HCV assembly competence of mouse liver cell lines.
This conclusion is supported by recent in vitro studies in mouse liver cell lines (22).
Nielsen et al. studied digitalized transmission electron micrographs (TEM) of mouse liver cell nuclei and compared normal liver, hyperplastic nodules and hepatocellular carcinomas [138].
Mouse liver cell line, Hepa1-6 cells were maintained in Dulbecco's modified Eagle's medium supplemented with 10% fetal bovine serum (FBS).
In vitro incubations of various mouse liver cell fractions with aflatoxin B1 showed that metabolites x1, x2, x3 and aflatoxin M1, could only be produced by the microsomal fraction and that NADPH was needed as a co-factor.
Previous proteome studies have compared primary mouse hepatocytes against a mouse liver cell line, 32 the dynamic phenotype of cultured rat hepatocytes, 33 or changes in the HepG2 proteome in response to hepatotoxins.
Similar(52)
The Stanford team put DNA, not RNA, into the mouse liver cells.
Both the AAV vector and the mRNA of Cas9 could be detected in the C3H mouse liver cells.
We analyzed the effect of differentiation stage of freshly isolated, mouse liver cells on the reprogramming efficiency.
Mouse liver cells then transcribed that DNA into RNA and spliced some of the corrected RNA into their own RNA.
And when the researchers simulated starvation in cultured mouse liver cells, the cells also ramped up SIRT1 production; when they added glucose, cells ramped it down.
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