We used the mouse genomic DNA sequence to test our ability to computationally predict 100 kb regions of the mouse genome that exhibit unusually short (<185 bp) nucleosome array periodicities, compared with the bulk chromatin periodicity of 195±5 bp, in mouse liver nuclei.
Proteins from mouse liver nuclei were prepared according to the NUN procedure (Lavery and Schibler, 1993).
Nielsen et al. studied digitalized transmission electron micrographs (TEM) of mouse liver cell nuclei and compared normal liver, hyperplastic nodules and hepatocellular carcinomas [138].
On the contrary, AhR protein accumulated in the nucleus but AhR-XRE complex decreased in the nucleus of ST-1 treated ST-1 treated
Hausp, Per2, Cry1, Cry2, Tubulin, and Lamin were measured by IB in whole cell lysates or nuclei prepared from mouse liver or quadriceps harvested at the indicated ZTs.
An interesting novel finding is the presence of CSE-positive nuclei in the hepatocytes of injured mouse liver.
In experiments with mouse liver tissue the mean intensities of NanoOrange were normalized to mean DAPI intensities for each nucleus.
Engineered vectors containing capsid variable regions (VR) VII & IX from AAV8 in an AAV2 backbone mediated near AAV8-like transduction in mouse liver, with higher numbers of chimeric genomes detected in whole liver cells and isolated nuclei.
The Stanford team put DNA, not RNA, into the mouse liver cells.
When they tested the animals' blood, they found breakdown products that are normally made by the human liver and not by the mouse liver.
