Sentence examples for mouse fibroblasts showed from inspiring English sources

Similarly, quiescent (PRE) mouse fibroblasts showed a robust c-Fos serum response, regardless of O2 concentration, whereas SEN XRA) mouse cells (in 3% O2) did not (Fig. 3D).

DNA methylation analysis by MassARRAY revealed that the miR-200c-positive keratinocytes showed minimal DNA methylation in the mir-200c CpG island, while the miR-200c-negative mouse fibroblasts showed extensive DNA methylation of all CpG sites in the region (Figure 6B).

Colonies formed from mouse fibroblasts showed the appearance of iPS cells.

Furthermore, studies using normal mouse fibroblasts showed that disrupting STAT3 signalling has much less profound effect in normal human and murine cells (Catlett-Falcone et al, 1999; Niu et al, 1999; Bowman et al, 2001; Burke et al, 2001; Song et al, 2005), suggesting that blocking STAT3 signalling may not be grossly toxic.

However, a study carried out by de Funari et al. [ 23] in 2007 on the effect of Brazilian green propolis on the viability of mouse fibroblasts showed that from 0.12 to 7.81 μg/mL, propolis revealed no statistically significant differences from the control, but concentrations of 31.25 μg/mL or more were toxic to the cells.

Moreover, some RAD51 paralog mutants show modest reductions in SCE, but isogenic rad51d mutant lines in both chinese hamster ovary and mouse fibroblasts show no decrease in spontaneous SCE [ 29, 30].

Recent studies on the senescence-associated secretory phenotype (SASP) of human and mouse fibroblasts show it is conserved across cell types and species and that specific secreted factors are strong candidates for stimulating malignant phenotypes in neighboring cells [ 22, 23].

Literature documents that diabetic mice fibroblasts show a severe impairment in VEGF production under normoxic and hypoxic conditions in addition to an increased pro-degradative activity due to the high expression of matrix metalloprotease type 9 (MMP-9) [ 28].

A recent study using Vps34-deficient mice and mouse embryonic fibroblasts showed that Vps34 does not control the basal activity of mTORC1, but is essential for acute amino acid-induced mTORC1 activation [ 31].

One group reported that the Psn-processed N-cadherin ICD targets CBP for degradation, and that Psn FAD-linked mutant mouse embryonic fibroblasts showed increased levels of CBP protein [14].

Moreover, Tln2cd/cd mouse embryo fibroblasts showed no obvious defects in cell adhesion, migration or proliferation.