Sentence examples for mouse models showed from inspiring English sources
Recent work in mouse models showed that the loss of p53 accelerated the development of medulloblastoma.
Results from experiments in SMA mouse models showed that ISIS-SMNRx effectively altered SMN2 mRNA splicing, increased SMN protein in the spinal cord, and had significant effects on functional and histological measures of neuromuscular health, including survival, when delivered to the CNS.
Work on mouse models showed that the immune system can only support a finite number of antibody producing cells [45], so it is also possible that EBV-induced expansion reduces the available niches for vaccine-specific antibody producing cells.
Ladiges et al. [14] recently reported that only 3 (Ames (Prop1df/df), Snell (Pit1dw/dw), growth hormone receptor knockout (GHR-KO)) out of 20 published long-lived mouse models showed consistent lifespan extension across separate studies.
Transmission studies in several wild-type or transgenic mouse models showed that these forms were associated with two distinct major strains of infectious agents, which also differed from the unique strain that had been isolated from cases of classical BSE during the food-borne epizootic disease.
Three mouse models showed deficits in the response probability during training.
Both mouse models show that TLX1 can induce T-ALL.
Since all circadian gene-mutant mouse models show increased sensitivity to γ-radiation, we conclude that the molecular clock functions in tumor suppression in vivo.
However, current humanized mouse models show sub-optimal human T cell reconstitution and limited ability to support immunoglobulin class switching by human B cells.
The combination of SS1P with chemotherapy is based on results from in vivo mouse models showing marked synergy between SS1P and chemotherapy [23] [26].
In vivo primary glioblastoma mouse models show efficacy.
