Sentence examples for mouse kidneys showed from inspiring English sources
Expression analyses in embryonic mouse kidneys showed that only Adi1 was moderately highly expressed.
Wholemount in-situ expression analyses of the mouse orthologs of these genes in embryonic mouse kidneys showed strong expression of Esrrg, encoding a nuclear steroid hormone receptor.
UOK257 xenograft tumors showed strong nuclear TFE3 staining whereas the adjacent mouse kidneys showed weak and diffused cytoplasmic or cytoplasmic/nuclear TFE3 staining (Fig. 4B, top right).
It is however known that leptin is mainly excreted by the kidney, and that rat and mouse kidneys show abundant Ob-R transcripts, indicating an interaction of leptin with special renal receptors [ 36, 37].
Immunohistochemistry analysis of normal mouse kidney showed that immunoreactivity was located on endothelial cells of glomerular capillary loops and peritubular capillaries.
Previous analysis of the developing mouse kidney showed NTRK2 to localize to the MM while in WT NTRK2 has been suggested as a bad prognostic marker(Durbeej et al. 1993).
The list of genes differentially regulated in control and diseased human kidneys only showed significant enrichment for X and Y chromosomes (Table 4), while the differentially expressed genes in the mouse kidney showed enrichment not only for sex chromosomes but for various autosomes (including chromosome 4, 7, 14, 19) (Table 4).
Studies on mouse kidney showed that of the 24 genes examined, several had variable expression through the different stages of renal development, whereas five were not expressed at all.
Thus, the regulatory activity of Tshz3 in the mouse kidney shows parallels with the activity of tsh in insect MpTs in regulating specialised renal cell differentiation.
Compared to 3 month-old young adult mice, aged kidneys showed a substantial decrease in capillaries, identified by CD31 staining, in both cortex and medulla.
In 8-month-old mice, KO but not KI kidneys showed tubular degeneration and extracellular deposition of lipofuscin (Fig. 1C and Supplementary Material, Fig. S3B and C).
