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Dr. Olney said his mouse studies showed that medical anesthetics could also cause the death of fetal neurons.
Both the phantom and in vivo mouse studies showed that superficial fluorescence signals could be imaged accurately.
We also included priming of the animals with mosquito salivary gland extracts (SGE), because recent mouse studies showed that this "natural" pretreatment may increase susceptibility to WNV [17].
However, recent mouse studies showed that no less than 10 out of 28 genes with putative cis eQTLs could not be confirmed by quantitative RT-PCR [4].
Subsequent mouse studies showed that deletion of the miR-17/92 miR-17/92s perinatal lethal.
Mouse studies showed that microglia from SOD1G93A ALS mice express higher levels of the prostaglandin E2 receptor (Di Giorgio et al, 2008; de Boer et al, 2014).
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According to Hermona Soreq, the head of Hebrew University's Institute of Life Sciences and a study author, the mouse studies show that "Pyridostigmine penetrates the brain rather effectively under stress conditions".
The APOE gene contains stretches of DNA that estrogen can bind, and mouse studies show the hormone can regulate APOE expression in a tissue-specific manner.
Clinical trials are in the early stages, and although dystrophin replacement looks feasible (mouse studies show we can put it back into tissue systemically), money for research is the determining factor on the time it will take to get a cure.
Recent mouse studies show that avidity for host cells can play an important role in polyclonal antibody escape, and further that electrostatic charge of the hemagglutinin glycoprotein can contribute to such avidity.
These findings are consistent with human and mouse studies showing that although loss of the second allele can accelerate tumor progression, mutation of a single LKB1 allele is by itself tumorigenic (i.e. LKB1 can be haploinsufficient) [9], [17].
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