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dC1GalT1 mutant larvae have also been shown to have morphological defects in the CNS.

Detection of cuticular chitin also reveals that the main tracheal branches, the dorsal trunks, of hau and gho mutant larvae have a much smaller lumen diameter than those of the wild-type (Figure 6A, C and E).

Here, we describe our finding that parkin mutant larvae have defective locomotion, with a bradykinesia-like phenotype.

buffy mutant larvae have unusual basal characteristics: increased Tor signaling, reduced energy source storage, reduced ATP levels and increased lactate levels.

Consistent with a previous report of a transient apoc2 knockdown, apoc2 mutant larvae have a minor delay in yolk consumption and angiogenesis.

Finally, we have found that odor receptor mutant larvae have behavioral phenotypes in response to odors that are weak electrophysiological ligands, consistent with the role of olfactory sensory neurons allowing gain control of the olfactory circuit.

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After aversive olfactory conditioning, dumb1 mutant larvae had significantly reduced scores compared to CantonS wild-type controls (Figure 4A; p = 0.013).

The cuticle of hau mutant larvae has a reduced epicuticle, and the chitin-protein organisation is lost in the procuticle, while the envelope has a normal appearance (Figure 2B).

However, a majority of itprsv35/ug3 mutant larvae had no or very little food in their gut and thus exhibit reduced absorbance values indicating that they were unable to feed normally (Figure 2A, B, 60 hrs after egg laying (AEL) and Figure 2C, D, 108 hrs AEL).

(H – H′ ) A btd mutant type II NB clone generated in pros 17 heterozygous mutant larvae has multiple mature INPs.

Dorsal ddaC neurons within zygotic BicD mutant larvae had a much less extensively branched dendritic arbor than wild-type cells.

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