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dlg mutant larvae display selective loss of glutamate receptors at the NMJ (Chen and Featherstone, 2005), and DLG protein is required for synaptic plasticity (Budnik et al., 1996; Thomas et al., 1997).
We previously reported that dC1GalT1 synthesizes T antigen expressed in hemocytes, lymph glands, and the central nervous system (CNS) and that dC1GalT1 mutant larvae display decreased numbers of circulating hemocytes and excessive differentiation of hematopoietic stem cells in lymph glands.
In fact, DLG mutant larvae display larger excitatory postsynaptic potential amplitudes [ 17].
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Interestingly, before dying, the fga mutant larvae exhibit defects in the process of gas filling of the respiratory (tracheal) system, a process that normally occurs by the end of embryogenesis [20].
Utx Δ mat- zyg- mutant larvae show complete loss of Ubx expression in a patchy pattern in haltere and third leg discs (Fig. 4C).
By the time that the larvae display motility problems (third instar) this difference is exacerbated, leaving very few clusters in the mutant situation.
Therefore we tested whether tbc-2 tm2241) tbc-2 tm2241ay reduced survivalarvae hatchedisplaye absence of food.
In gho mutant larvae traces of shallow taenidia can be distinguished at the ultrastructural level (Figure 6F).
Initially, Manf Δ96 mutant larvae hatch and feed normally.
Drosophila larvae display defecation cycles of stereotypic frequency, involving sequential contraction of hindgut and anal sphincter.
During this time, larvae display negative phototactic behavior that is mediated by a ring of long-ciliated, pigmented cells at the posterior pole [ 14].
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