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We analyzed the molecular mechanism underlying androgen-induced cell migration in mouse NIH3T3 fibroblasts.
These studies have defined the pattern of cranial neural crest cell migration in mouse embryos and demonstrated that the frontal bone is of neural crest origin, whereas the parietal bone is of mesoderm origin.
These data indicate that fascin 1 not only promotes cell migration in mouse melanocytes but it also has a role in growth and cell cycle progression.
Accordingly, in-vivo observations of leukocyte rolling and migration in mouse retina now accurately evaluate the genetic regulation of leukocyte behaviour in the inflammatory response.
Pdgfra is important for oligodendrocyte maturation and migration in mouse (McKinnon et al., 2005), although the function of pdgfra in zebrafish oligodendrocyte development has not been tested.
Consistent with this notion, recent studies have shown an effect of anti-CD97 treatment on cell migration in mouse models of infection and inflammation.
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Mutations in Tuba1 cause abnormal neuronal migration in mice and lissencephaly in humans [21].
Here, we examined NC migration in mice lacking Vangl1/2 function.
Moreover, inhibition of CCR9 reduced arthritis and inflammatory cell migration in mice.
Canonical Wnt/β-catenin signalling is known to be required for NC migration in mice (Ikeya et al., 1997).
In addition, since Lis1 activity is sufficient to promote bipolar migration in mice, over-expression of DCX in this organism will have no effect on bipolar migration.
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