Sentence examples for mice we made from inspiring English sources

In serum from K14-HPV16 transgeniCstc+/+/+ and Cstc−/− mice, we made similar observations.

Therefore, in chronic imaging experiments in adult mice, we made quantitative measurements of the retinotopic maps before and after enucleation.

To study the function of Notch signaling in the skin of adult mice, we made use of a series of conditional gene targeted mice that allow inactivation of several components of the Notch signaling pathway specifically in the skin.

In the course of characterizing of the properties of mouse embryonic fibroblasts (MEFs) derived from wild-type and CYLD−/− mice, we made the surprising observation that the CYLD−/− MEFs had elevated proliferation rate compared to the CYLD+/+ in the presence of serum.

Nevertheless, in control experiments analyzing pubertal MMTVrtTA/H2BGFP mice, we made the unexpected discovery that H2BGFP was expressed primarily within the CD24+/CD29+ and CD24+/CD29lo compartments, which contain mammary stem cells and progenitors, respectively.

Before evaluating the immune responses induced by them in a mouse model, we made sure that: (i) the sizes of the two OVA-nanoparticles did not extensively overlap, (ii) the nanoparticles have similar zeta potentials and comparable antigen-loading, and (iii) the nanoparticles did not aggregate when suspended in simulated biological media.

Furthermore, to perform in vivo cell lineage tracing in both mouse models, we made use of the ROSA26 Cre reporter line (R26R) [ 27], which expresses β-galactosidase (β-gal) upon Cre-mediated recombination.

Because we did not possess an antibody to the mouse LG domain, we made use of transgenic mice with doxycycline-inducible expression of human laminin α5, which assembles with mouse laminin β2 and γ1 to generate a functional chimeric human/mouse LM-521 trimer (Goldberg et al., 2010).

Although the IFN α effects on stem cells are only demonstrated in mice, we here make the assumption that IFN α acts similarly in humans.

However, as the mean±SD body weight of MBL-null mice (26.50±0.46 g) was smaller than that of WT mice (29.05±0.93 g) (p<0.001), we made some calculations to reassure that the reduction in body size in the MBL-null mice did not contribute to the differences in infarct volume vs. WT.

Using TE-Array to profile transposon expression in a variety of normal mouse tissues, we make two encompassing observations.