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During swimming V1 IN-ablated mice show extensive co-activation of the GS and TA muscles.
As anticipated, Calca -/- mice show extensive osteolysis compared with wild-type mice, and CT substitution reduces particle-induced osteolysis.
These mice show extensive vacuolization in epithelial cells in the seminiferous tubules which might be caused by accumulation of TGs and CEs.
Interestingly, when infected with scrapie these mice show extensive PrPSc depositions in the form of amyloid plaques, rather than the usual non-amyloid form of PrPSc, but lack clinical signs [ 5].
KL-deficient mice show extensive and accelerated arteriosclerosis in association with medial calcification of the aorta and both medial calcification and intimal thickening of medium-sized muscular arteries [ 26].
During our studies on the IRF family of transcription factors in the context of regulation of PRR signaling and oncogenesis, we observed that mice genetically deficient for IRF5 (IRF5-deficient mice) show extensive lung metastasis of the B16F1 melanoma cells (hereafter referred to as B16 cells), a consequence reported to be controlled by NK cells.
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As revealed by Sirius red staining and hematoxylin-eosin (H&E) staining evaluation, we found that TCDD-exposed mice showed extensive disruption of liver architecture, including hepatocellular necrosis, inflammatory cell infiltration, and fibrosis.
Electron microscopy of glomerulus from transgenic mice showed extensive podocyte foot process effacement.
Finally, electron microscopy of glomeruli from transgenic mice showed extensive podocyte foot process effacement.
Expectedly TLR2/9 double knock-out mice showed extensive susceptibility to Mtb infection.
Of note, cochlear tissues of Cmah-null mice showed extensive apoptosis, whereas control mouse showed a moderate apoptosis.
Related(20)
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