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To clarify how cell size contributes to leaf elongation in slr1 and pla1/slr1 double mutants, we compared the lengths of epidermal cells on the adaxial side of the third leaf sheath in each mutant (Figure 2A).
To gain further insight into the encapsulation defects of mysnj42 and Rac1J11 mutants, we compared the ability of control and homozygous mutant haemocytes to adhere to wasp eggs.
To investigate the reduction in IPP binding affinity for the FPPS mutants, we compared structures of wtFPPS and mutant FPPS.
To test the robustness of gene expression in this set of hormone mutants, we compared two different growth conditions.
To determine if this was true for ND2 mutants, we compared the lifespan of ND2 mutants and controls.
For each of the 6 EMS-treated colonies (putative mutants), we compared the morph distribution following photoperiodic treatment to the wild-type (P212 clone) distribution.
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To investigate whether a reduction in vegetative growth rate or prolonged plastochron (i.e. the time between one leaf initiation and the next) causes the late-flowering phenotype in the z3 mutant, we compared leaf emergence rates between the wild type and the z3 mutant under long-day conditions (14.5 h-light, 30 °C/9.5 h-dark, 24 °C) in the growth chambers.
For each mutant we compared the binding competition of labeld synthetic (vector) RNA with unlabeled cellular RNA.
To identify the biochemical and metabolic pathways that are altered in the liver of XpdTTD mutant, we compared the gene expression profiles between XpdTTD mutant and wild type.
Since the synthesis and secretion of LcrV is reduced in the ΔrelA ΔspoT mutant, we compared production of three cytokines (IL-10, INF-γ and TNF-α) in mice infected with Y. pestis KIM5+ and χ10004 pCD1Ap).
To determine the morphological defects of the cotton GMS mutant, we compared the anthers of the mutant and its fertile wild type (WT).
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