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Exact(6)
Glucose although could cause root hair initiation in auxin signaling mutant at high concentrations but root hair elongation was severely compromised (Figure 9) suggesting the requirement of normal auxin physiology a prerequisite even for this glucose induced parameter of root growth and development.
Genes encoding the PTH11-type receptors were significantly more abundant among genes that are higher expressed in the Δlae1 mutant at high growth rates.
Analysis of D311E lipase crystal structure revealed an additional ion pair around E311 that may regulate the stability of this mutant at high temperatures [ 94].
Furthermore, the double mutant resembled the tph-1 mutant at low bacterial levels and the daf-7 mutant at high bacterial levels, suggesting that these genes act in parallel rather than in a linear pathway.
Cell cycle profiling of this diploid mutant at high (μM) concentrations of inhibitor therefore allowed us to probe Cdc28p activities lower than could be obtained in the tetraploid series (as discussed above), and confirmed the model predictions.
Because the lethality of the erg26 mutant at high temperature is linked to the accumulation of toxic sterols, we speculate that the incorporation of toxic sterols into the mitochondrial membrane could upset its sterol to lipid ratio and that HU can alleviate such damage to the mitochondrial membrane thereby reducing petite frequency.
Similar(54)
Most diagnostic is a distinct three-band pattern observed in the mutant at higher energy, which is very different from wt.
Recent advances in recombineering using synthetic oligonucleotides enable the rapid generation of mutants at high efficiency and specificity and can be implemented at the genome scale.
The sequence analysis of these genes revealed that most represent cells of distinct B-cell clones which expanded locally, generating somatic antibody mutants at high rate.
Additionally, the transcripts of certain genes for chlorophyll biosynthesis, photosynthesis and chloroplast development were severely affected in tcm5 mutants at high temperatures.
In addition, the impaired chloroplasts (Fig. 2b) and the abnormal transcripts of genes for chloroplast development (Fig. 7) were observed in tcm5 mutants at high temperatures, and those affected genes under high temperatures, at least, in part could recover to the comparable levels to WT plants at low temperatures (Fig. 8).
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