Sentence examples for modulation of fetal from inspiring English sources

For example, modulation of fetal oxidative stress has been reported after maternal glucocorticoid administration in both experimental [32, 33] and clinical settings [34 36], but it remains unclear if these effects vary depending on the presence or absence of inflammation.

We postulate from these findings that TGF- β1 may play a part in the modulation of fetal growth.

Most of the clinical studies published to date have evaluated total cord blood adiponectin (cbAdiponectin) in the peripartum period, so it has not been possible to establish a long-term modulation of fetal adiponectin production by mAdiponectin levels.

The fact that levels were significantly higher in amniotic fluid of preeclamptics [ 20] would provide further support to a possible role for TGF- β in the modulation of fetal growth.

Thus, our data would suggest that mAdiponectin early in the third trimester may be one of the factors implicated in the modulation of fetal adiponectin secretion, and that the multimeric forms and their distribution are the most implicated.

Our objective was to evaluate the role of parity in the modulation of the fetal programming of growth and vascular responses in these transgenic mice.

This suggests that modulation of GLI3 in the fetal placenta, and perhaps in other fetal tissues, contributes to arsenic's detrimental effects on fetal growth.

We hypothesized that macrophages in the pregnant endometrium would express genes that support placental growth (angiogenesis and tissue remodeling) and that are indicative of a M2 phenotype that signifies a role in modulation of immune responses towards fetal antigens.

Similar expression of miR-483-3p miR-483-3p miR-483-3pns suggests that modulatinn of miR-483-3p bothooratstandnutrition is a conserved phumansnon.

Although the present experimental design did not allow us to differentiate between direct effects of EDCs at the level of the pituitary and indirect central actions, the results clearly demonstrate that maternal EDC exposure could affect fetal intrapituitary modulation of LH release.

Sequelae such as central nervous system damage and sensori-neural hearing loss result more directly from fetal cytopathic injury, although modulation of placental immune function may also play a role in mediating fetal injury [ 33].