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Rats were infected with L. monocytogenes and NK cells from the spleen and bone marrow were analyzed 24 or 48 hrs p.i.
Mice were sacrificed within 0 12 hours of birth, and single cell suspensions from thymus, spleen, and bone marrow were analyzed by flow cytometry.
In this study, DNA and RNA from both blood and bone marrow were analyzed and 16% were found to display ASE [ 7].
Following hematopoietic reconstitution, the spleen, thymus, and bone marrow were analyzed for the presence of GFP+ and Cherry+ fluorescence markers in specific hematopoietic lineages by flow cytometry.
Methods: Mice were exposed to HEPA-filtered air or concentrated ambient fine particulate matter (CAP, 30 100 µg/m) from downtown Louisville Kentuckyy) air, and progenitor cells from peripheral blood or bone marrow were analyzed by flow cytometry or by culture ex vivo.
Similar(55)
Bone marrow was analyzed for the presence of apoptotic cells by M30 staining.
The nature of the NK cell defect in the premature infant was not defined, and bone marrow was analyzed with no pathologic findings noted.
After 4 weeks, bone marrow was analyzed to assess pro-B-cell levels, cytoplasmic λ5, and responses to TNFα in vitro.
Furthermore, in the total of 125 patients whose bone marrows were analyzed before and after rituximab therapy, there was no change in the bone marrow CD4/CD8 ratios (mean 1.2 at both time points).
Bone marrow chimeras were analyzed at 4 8 weeks post-bone marrow transfer (BMT).
Homozygous mutant ΔHSs bone marrow cells were analyzed in a similar fashion, but only Ly9.1+ cells were included in the analysis.
Related(20)
bone were analyzed
marrow were evaluated
marrow were assessed
marrow were analysed
marrow were tested
marrow were investigated
cord were analyzed
marrow were examined
brain were analyzed
marrow were characterized
marrow were generated
marrow were superseded
marrow were used
marrow were cultured
marrow were assumed
marrow were bombed
marrow were incubated
marrow were estimated
marrow were exposed
marrow were measured
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