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The intermediate levels of LDH release by both point mutants were partially inhibited by the addition of cytochalasin D (black bars, Fig. 6C).

Although the maximal effect for different cell lines was achieved at different concentrations of glucosamine and at different treatment time (not shown), for 8 of 9 cell lines a statistically significant increase in the activity was observed (Figure 5C), suggesting that some of the mutants were partially stabilized by the glucosamine, correctly processed and targeted to the lysosomal compartment.

Four additional Fix- mutants were partially analysed.

Selected Fis mutants were partially purified and assayed for activation of Hin-catalyzed inversion in vitro.

Both ΔUBA and ΔUIM truncation mutants were partially defective in ubiquitin-binding.

The aberrant morphologies of the colonies of Cafig1Δ, Cacna1Δ, and Cacnb1Δ mutants were partially alleviated on supplementation with exogenous Ca2+, supporting the view that these genes are involved in calcium signaling in C. albicans.

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Similar(53)

The results showed that yellow-green phenotype of egy1 mutants was partially restored by glucose treatment (Fig. 7a).

In addition, our results showed that the yellow-green phenotype, chlorophyll content and ion leakage of egy1 mutants was partially restored after exogenously applied glucose for 5 weeks.

Furthermore, the yellow-green phenotype of egy1 mutants was partially restored by 2 % glucose (Fig. 7), suggesting that the symptom of early senescence was relieved after glucose supplementation.

Compared to wildtype cells, ubp2Δ mutants are partially sensitive to the drug.

The premature lethality of yata mutants was partially but definitely rescued by the neuronal expression of yata.

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