Since the ΔclpK mutant was incompletely complemented by pClpK, a polar effect due to the deletion was considered.
In acs-20 mutants, the alae were incompletely extended compared to those of wild-type animals (Fig. 4C and D, black arrows), and unidentified matrix that was rarely observed in wild-type accumulated within the cuticle layers (Fig. 4E and F, arrowheads).
In contrast, the ACE mutants W594R, R828H, Q1136X and G1145AX were incompletely glycosylated.
Even in the complete absence of neurexin (Df 3R Exel6191), NMJ synapses appeared relatively normal, except as also noted in larval neurexin mutant NMJs [37], pre and postsynaptic densities were incompletely apposed in the absence of neurexin.
These data suggest that all the mutants, except R1180P and R762X found in the culture medium, were incompletely glycosylated and retained in the ER.
The EFI was not calculated, and complications were incompletely reported.
Outcomes were incompletely reported; there was high risk of bias.
In pha-2 mutants, the pharynx is incompletely elongated, exhibiting a shortened pharyngeal isthmus as a result of defects in the differentiation of the pm5 muscles (Avery 1993; Morck et al. 2004).
In previous studies, we have noted several instances in which mutant or wild-type ATP synthase was incompletely inhibited by inhibitors like fluoroaluminate, fluoroscandium, sodium azide, NBD-Cl, polyphenols, and peptides.
However, the seu and lug single mutants display a relatively mild disruption of ovule integument development that is incompletely penetrant.
