Sentence examples for mutants we analysed from inspiring English sources

To determine if any residual or fused Hsp90α protein persists in gene disruption mutants, we analysed the same tissues from homozygous mutant mice.

In the subset of mutants we analysed we found a total of 38 new WD40 repeat units.

The study by Changolkar et al. was performed using a fixed mutation on the C57Bl6 background whereas the mutants we analysed had a genetic background composed of half 129Ola and half C57Bl6 (intercrosses of the F2 generation).

To investigate the contribution of the failure in phagosome processing to lamellipodial and motility defects in SCAR Δ37 mutants we analysed SCAR Δ37 ;H99 double mutants, which lack all apoptosis.

To characterize the glycosylation state of WT ACE and the mutants, we analysed ACE in cell lysates and culture medium after treatment with either PNGase F (peptide: N-glycosidase F) or Endo H (endoglycosidase H).

The second cross was done in the same way, but with fewer lines: for the ceob2 mutant, we analysed three plates each of three independent lines; for the trt-1 mutant, we analysed eight plates of one line; and for the trt-1/ceob2 mutant, we analysed eight plates each of three different lines.

To examine the extent of fluorophore labelling on the V216C mutant, we analysed the mass values of the V216C mutant before and after labelling with fluorescein-5-maleimide using ESI MS.

To further dissect the phenotype observed in the cenp-metaΔ single mutant, we analysed living cenp-metaΔ neuroblasts by time-lapse microscopy to determine the mitotic timing (i.e. the time that elapses between Nuclear Envelope Breakdown (NEB) and anaphase onset).

As for the dag1gai-t6 double mutant, we analysed by PCR-based genotyping more than one hundred F2 plants derived from both the dag1 × gai-t6 and the reciprocal cross, but we were unable to isolate the dag1gai-t6 double mutant.

Considering the large number of mutants we have analysed, this would suggest that PSbMV infection is dependent on a rather limited number of eIF4E residues in defined positions, although we acknowledge that members of the R/− group may also be involved.

To further evaluate the phenotypic consequences of mitochondrial abnormalities in ref(2 P mutants, we next analysed their overall survival.