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The resulting spectrum showed no obvious change between wild-type and mutant proteins, indicating that the overall folding of the mutants was the same as the wild-type proteins (Fig. S2).

One difference between the two pmt4 mutants was the method of gene mutation.

The null hypothesis was that the proportion of mutants was the same in both sources of sequences (NCBI vs. Ottenby).

Underlying thethermoresistance phenotype of atx-3 mutants was the significant up-regulation of several molecular chaperones, namely HSP-1, HSP-16.1, HSP-16.2, HSP-16.49 HSP-16.4944E5.5, C12C8.1 and DAF-21.

With the exception of QacR E90A, which displayed double the basal induction level, the basal level of expression from PqacA for the QacR mutants was the same as for wt (Table 2), indicating that DNA-binding was not impaired by these mutations in the QacR multidrug-binding pocket.

The only significant difference phenotypically between the two mutants was the higher penetrance of abnormal teeth on the mandible in Wise mutants, which we attribute to the fact that the Lrp4 mutation is hypomorphic, whereas Wise is believed to be a null.

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The New Mutants are the outcasts.

Magneto is a powerful mutant who had rejected society, believing that the outsiders and humans can never coexist and that mutants are the heirs to the world's future.

This would be meaningless if the killed mutants are the same.

The most striking feature of atips1 mutants is the spontaneous lesion formation on leaves.

The 05342 and 04624 mutants were the next strongest by this measure.

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