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Exact(7)
Three of the mutants presented a conditional-lethal phenotype in vitro.
However, 10 of the mutants (designated AM 1 10, meaning Adhesion or invasion defective Mutants) presented a higher than 50 % reduction in adherence in comparison to the adherence observed in the parent strain (Fig. 2).
Cytological analysis revealed that esk1 mutants presented a strong irx phenotype in roots, hypocotyls and stems (Figure 6).
Upon IPTG induction, both SmpB and SsrA conditional mutants presented a slightly higher growth rate when compared with the wild type strain (for SmpB, see Fig. 2).
The cortex of Pafah1b1+/−;Vldlr−/− double mutants also appear fairly normal, however, that of Pafah1b1+/− Apoer2−/− double mutants presented a severe abnormality similar to the cortical layer inversion typically seen in Reln−/−, Dab1−/− or Apoer2−/−;Vldlr−/− mutants (Fig. 5).
Furthermore, all mutants presented a particular set of active TNF-R1 downstream pathways.
Similar(53)
Unexpectedly, the other class of Bug22 mutants presented an even more severe phenotype.
Because of their large numbers, embryo-defective mutants present a special challenge for saturation mutagenesis.
The emergence of HBsAg mutants presents a challenge to HBV screening programs.
In S. cerevisiae, cda1 mutants present a more diffuse chitosan layer, while their surface layer remains intact.
Arabidopsis thaliana drb2 mutants present a 2 to 10 fold increase in RdDM associated p4-siRNA ([ 50] and Figure 1a) again suggesting that plant DRB2 is involved in regulating small RNA biogenesis and is therefore a regulator of RdDM.
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