Sentence examples for mutant is blocked from inspiring English sources

However, in the state of SAR, their biological functions are both redundant and essential, since only the tga2 triplega6 triple mutant is blocked from induction of PR1 during SAR (Zhang et al. 2003).

The fact that minor perturbation of final insulin structure caused by G(B23)V substitution is greatly outweighed by major defects in the ability to assemble the insulin chains with proper cysteine pairings indicates that this MIDY mutant is blocked primarily in the protein folding pathway rather than in the stability of the insulin native state.

These results indicate that the folA1 mutant is blocked in M phase at the restrictive temperature.

This suggests that the Δ csp-2 mutant is blocked at the double-doublet stage before connective formation takes place.

The kar5-C68A mutant is blocked with wide bridges, suggesting that this mutation must block downstream of dilation.

Kinesin activity was inhibited with aurintricarboxylic acid (ATA), which impeded replication of wild-type and the sipA ++ strain, and although both remained perinuclear, peripheral migration of the sipA − mutant was blocked.

We show here that the negative action of UNC-2/ UNC-36 at the M4 neuromuscular junction requires SLO-1 and that the increased TB pumping seen in unc-2 mutants is blocked by a mutation in the L-type voltage-gated calcium channel EGL-19.

Further analysis of mutants (R18A, I141A, L143A and E153A) synapse and cleavage showed that these mutants were blocked in recombination at the stage of strand cleavage.

The chosen cpd, ga1, and aba2 mutants are blocked in the biosynthesis of BRs, GAs, or ABA, respectively (Fig. 1), and each exhibits deficiency phenotypes specific for the respective hormone.

Activation of NFAT by TRPC6 mutants was blocked by inhibitors of calcineurin, calmodulin-dependent kinase II, and phosphatidylinositol 3-kinase thus identifying the activation of the calcineurin-NFAT pathway as a potential mediator of FSGS.

Again, CME in clathrin-depleted cells expressing these mutants was blocked by Pitstop 2 (Fig. 3).