Sentence examples for movement abnormalities in from inspiring English sources

The commonness of visual sensory and eye movement abnormalities in MS highlights the importance of understanding the principles addressed in this review.

Gait analysis is a widely used method to obtain objective data in 3D in the clinic in order to detect movement abnormalities in both children and adults.

These factors are, however, linked significantly with the other NSS cluster-movement coordination abnormalities-which may reflect different neurobiology and etiology of individual movement abnormalities in schizophrenia.

In contrast to informal clinical evaluations of oculomotor dysfunction frequency (previous studies: 38%, current clinical examination: 33%), detailed eyetracking investigations revealed eye movement abnormalities in 80% of patients with posterior cortical atrophy (compared to 17% typical Alzheimer's disease, 5% controls).

Also, the progression of Mn-induced parkinsonism appears to be a gait disorder of early onset with dystonia that occurs much later in the slow progression of the movement abnormalities in PD.

Autonomic seizures are a consideration in sudden infant death since recurrent episodes of apnea, reported in infants who subsequently die of SIDS [ 47], may be the sole manifestation of seizures (without movement abnormalities) in infants with temporal lobe pathology [ 36].

To further test the connection between loss of GALT enzyme activity and the movement abnormality in dGALT ΔAP2 homozygotes, we restored GALT activity in these animals using a human GALT transgene driven by the GAL4/UAS system.

The impaired ability to sequence movements was the most frequent movement abnormality in schizophrenia patients: 67% of the patients had at least one mild impairment of movement sequencing as compared to a 33% incidence of sensory integration problems, and 13% of motor coordination abnormalities.

Simply put, our current understanding is that a primary defect in biochemistry (GALT deficiency) leads to physiological changes that are either localized or systemic, and that these physiological changes ultimately lead to impaired neuronal or neuromuscular function and a movement abnormality in the mutant flies.

In fact, the new evidence suggests that it may be the dysfunction of this system or the inability to release dopamine that produces the movement abnormalities documented in Mn-exposed subjects.

An important observation on the etiological role of Mn in the parkinsonism in these young addicts is based on the fact that movement abnormalities are observed in ephedrone users in Eastern Europe and Russia where the chemical preparation uses potassium permanganate as the oxidizing agent.