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The model predicts that the concentrations of diffusible extracellular Aβ species in the agarose are close to the concentrations in brain tissue after ∼30 hours and the mouse was therefore left in its home cage for ∼60 hours.
The set of primary sequences for each mouse was therefore mapped into a numeric feature vector of length 100.
A urothelially restricted NGF-OE mouse was therefore created to study what happens to the micturition phenotype [ 94].
Despite the transgenic Vβ12 chain being expressed on virtually all CD3+ T cells, however, it may still recombine freely with endogenously derived α-chains, and the frequency of CII-specific T cells in the Vβ12-tg mouse was therefore not known.
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The mouse is therefore not the only cancer model that has been used for cross-species comparison.
The Cd2ap mutant mouse is therefore an excellent model system for the study of podocyte dysfunction driven glomerulosclerosis.
The mouse is therefore an excellent model organism to investigate the fundamental events driving dental development and for studying the molecular pathogenesis of AI.
The chromosomally engineered Df1/+ mouse is therefore a good model for the human syndrome and provided the opportunity for gene targeting and transgenic complementation experiments.
Since HPB regimen showed higher IL-4 after vaccination, and substantially lower IL-4 after challenge infection, early mixed Th1/Th2 responses exhibited by this groups of mice was therefore skewed in Th1 biased response after L. donovani infection.
The finding of proteinaceous aggregates containing SOD1 in motor neurons of postmortem fALS patients and transgenic mice was therefore a major advance in the field since it suggested that aggregation of SOD1 is related to the pathology of SOD1-linked fALS [10].
The ability of SRTAW04 to attenuate neuronal loss in MHV-A59 infected mice was therefore examined.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com