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These mouse studies indicate that dysregulation of postmeiotic repression can lead to male subfertility or infertility.
Mouse studies indicate that either is sufficient for melanoma predisposition and that they might, in fact, cooperate to drive disease progression [76] [77].
Transgenic mouse studies indicate that DAT is critical in maintaining normal dopaminergic neurotransmission and synaptic tone, and that even a 50% reduction in DAT protein (DAT +/− mice) is sufficient to significantly alter DA signaling and synaptic stores [6], [7].
While there are no known human disorders caused by defects in RECQ1 or RECQ5, cellular and transgenic mouse studies indicate that loss of these proteins also leads to genomic instability [5].
Parallel mouse studies indicate that the main effects are due to nab-paclitaxel and not gemcitabine.
In addition, model mouse studies indicate that lipid transport by ABCA12 is indispensable for intact differentiation of keratinocytes.
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Analysis from budding yeast, fly, and mouse studies indicates H4S1ph may be involved in gamete-associated packaging of chromatin [ 9].
In multiple sclerosis biopsies, Ptprz1 is expressed in lesion areas with cells expressing PLP, which together with the mouse studies indicates a potential role of Ptprz1 in oligodendrocyte survival and remyelination in demyelinating diseases (Harroch et al., 2002).
The results from this mouse study indicate that the immune-selective action of dipyridamole may not amplify the osteoporotic effect and fracture risk associated with very-low-dose prednisolone [ 48] in the clinical setting with chronic dosing.
Now, a new mouse study indicates that the belief that a mother's diet can influence a baby's sex is not so far-fetched.
In conclusion, our mouse study indicates that proximal tubular reabsorption of urine hepcidin-1 occurs in a megalin-dependent manner.
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