Sentence examples for mouse data indicate from inspiring English sources

Clinical and in vivo mouse data indicate that HB-EGF drives tumor progression [ 17- 19].

3) The manuscript argues that the germ-free mouse data indicate that there is an endogenous antigen-driven selection program at work, but does not rule out food antigens as sources for this selection.

Our mouse data indicated control F1 mean ages at VP were 27.0 (Tyl et al. 2008b) and 29.4 (Tyl et al. 2008a) days, with no phytoestrogen effect, because Thigpen et al. (2003) reported the following percentages of females acquiring VP at 22 30 days of age: for low D&G diets (0 20 μg/g), 3.6 86.6%; for midrange D&G diets (101 210 μg/g), 23.8 87.7%; and for high D&G diets (270 370 μg/g), 37.5 96.0%.

In addition, the knockout mice data indicate that the DLC members might play different physiological roles in development.

The Salm3−/− mice data indicate that SALM3 is important for excitatory synapse development in the hippocampal CA1 region, as supported by substantial reductions in mEPSC frequency (~52.3%) and PSD density (~14.5%).

Interestingly, our mouse tumor data indicate that the RGD domain of OPN mediates tumor growth and metastasis mainly through decreased apoptosis.

Finally, our preliminary analyses of mouse expression data indicate a very similar trend (data not shown) as with human data.

10.7554/eLife.03058.005 Figure 3. Human and mouse ENCODE data indicate that Mdgt −/− lines contain deletions of conserved binding sites for transcription factors and chromatin regulatory proteins.

Because hematopoietic cells from young mice failed to mature properly in the bone marrow of aged mice, these data indicate a deficiency in the stromal cells of aged mice.

Together with the premature death of Col1a1 r/r mice, these data indicate that reduced type I collagen catabolism imparts a progeroid phenotype in mice.

Although MCP-1, TNF-α, IL-17A and IFN-γ as well as joint inflammation were suppressed in the early phase of arthritis in IL-23p19KO compared to WT mice, these data indicate that the early joint inflammatory responses in this non-autoimmune T cell mediated arthritis-model is at least partly IL-23 independent.