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Contrast enhancement on image studies indicate chronic inflammation and partly hypointense areas within the Teflon felt on T2-weighted MRI-scans or hyperdense areas on CT-scans may indicate calcification [24].
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Subsequent image studies indicated insufficient healing progression with bone deformity.
Imaging studies indicate that monoamine reuptake inhibition by amphetamine and trace amines is site specific and depends upon the presence of co-localization in the associated monoamine neurons.
Imaging studies indicate a link between degeneration of the intervertebral disc (IVD) and LBP [ 3, 4].
Imaging studies indicate a link between IVD degeneration and LBP [ 3, 4], with the most clinically significant correlations between degenerate disc space narrowing (which develops as degeneration progresses [ 5, 6]) and chronic LBP [ 7].
Imaging studies indicate that chemotherapy seems to particularly affect the integrity [71] of the white matter with several studies demonstrating a link between the abnormal microstructural properties of specific white matter regions and cognitive impairments seen in patients treated with chemotherapeutic agents [72].
Imaging studies indicated pituitary apoplexy with a large hypodense pituitary lesion with widening of the sella turcica.
Imaging studies indicated that GPR91 is located specifically on the plasma membrane, and is internalized and then desensitized as a result of ligand stimulation [11; reviewed in 12].
Imaging study indicates that the tumor was classified as type IIIB HCCA according to the Bismuth-Corlette classification system (data not shown) [8].
While men do consider androgenetic alopecia an unwanted and stressful event that diminishes their body image satisfaction (studies indicate 50% of men with mild hair loss and 75% with moderate to severe hair loss report concern [ Cash 1992]), society tends to regard hair loss in men as expected and normal due to the greater visibility of hair loss within men.
However, oxidative stress-induced mitochondrial dysfunction likely alters glucose metabolism and consequently impairs neuronal function in MS. Imaging and pathological studies indicate that glucose metabolism is altered in MS, although the underlying mechanisms and its role in neurodegeneration remain elusive.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com