Sentence examples for mouse skin resulted from inspiring English sources

Previously, it was shown that targeted activation of the Rho effector ROCK2 kinase to mouse skin resulted in increased collagen deposition [ 47].

From our analyses, it is ubiquitous that application of DMBA/TPA on mouse skin resulted in activation of NF-κB, as similarly reported previously [ 41].

Topical application of TPA on mouse skin resulted in discernible oxidative response as measured by increased hydrogen peroxide and MDA production and depletion of antioxidant defense like catalase [ 8] and GSH.

The ectopic expression of miR-203 in the basal layer of mouse skin resulted in perinatal lethality, which was associated with a lack of epidermal basal progenitors and the detection of sporadic ΔNp63-expressing cells.

Further, treatment of EGCG to mouse skin resulted in marked inhibition of a single UVB induced phosphorylation of ERK1/2 (16 95%), JNK (46 100%), and p38 (100%) proteins in a time-dependent manner.

Pomegranate seed oil (5%) and PFE application prior to TPA on mouse skin resulted in delay in latency period from 9 to 14 weeks and afforded considerable protection in terms of tumor incidence and tumor multiplicity [ 103, 104].

Thus, NB-UVB irradiation of imiquimod-treated mouse skin results in improvement of psoriasiform dermatitis, as assayed by clinical, histological and immunohistochemical parameters.

Unlike wild-type SMO, expression of SmoM2, an activated SMO mutant identified in human BCCs, in mouse skin results in the formation of BCC-like tumors [107].

Similarly, loss of Dsp in mouse skin results in epithelium peeling that is more pronounced within the basal and spinous layers, leaving large areas of denuded skin after mild mechanical stress (38).

The ectopic expression of miR-24 in the basal layer of the mouse skin results in the premature expression of differentiation markers, and an early exit from the cell cycle.

In our previous study, it has been shown that postnatal overexpression of the LMNA c.1824C>T mutation in mice skin results in increased proliferation and premature senescence (Rosengardten et al., 2011).