Microinjection of fluorocitrate into the right striatum resulted in a significant decrease of the uptake of both [14C]phenylacetate and [14C]acetate into the right striatum.
It also unambiguously confirms the degree of segregation of MSNs in the mouse striatum and allows the full exploitation of results obtained with BAC-transgenic mice.
Moreover, results indicate strong contribution of genetic background to differences in gene transcription in the mouse striatum.
Specific binding of 123I-β-CIT within the mouse striatum could be clearly visualized with SPECT.
Following grafting into the mouse striatum, adult human astrocytes survived and expressed the transgene for at least 8 weeks.
We surmise that aging contributes to the cellular and molecular architecture in the mouse striatum with implications for neurodegeneration.
When transplanted into the mouse striatum, Pitx3-expressing cells generated an increased proportion of AHD2-expressing DA neurons.
Neural stem cells derived from hES cells using ES-ACM were implanted into the mouse striatum.
NS were originally derived from the embryonic and adult mouse striatum subventricular zone.
Thus, the LV-Crym-HA was efficacious to increase Crym expression in the mouse striatum.
