Sentence examples for models for malaria from inspiring English sources

Non-human primate (NHP) models for malaria are admittedly under-used, although they are probably closer models than mice for human malaria; in particular, NHP models allow the use of human pathogens (Plasmodium falciparum, Plasmodium vivax, Plasmodium malariae and Plasmodium knowlesi).

These results mirror those found more generally for the range of infectious diseases in which endemic stability is predicted to occur as well as in simpler models for malaria immunity [18], [19], [25] [27].

We have observed similar, though smaller, effects with simulations of vaccination using the same models for malaria in humans (Maire et al. 2006).

The models for malaria in humans (Smith et al. 2006, 2008) have already been used to investigate the effects of vaccines (Penny et al. 2008; Smith et al. 2012) and intermittent preventive treatment (Ross et al. 2008, 2011) in reducing malaria morbidity and mortality.

The Poisson models for malaria were further restricted to camps with an average monthly malaria incidence rate of 4 or more cases/1000 under five population/month in a given year to exclude camps in regions where malaria transmission was minimal or absent.

A model for malaria interventions is at risk as funding flattens and the disease smolders on.

We present a model for malaria dynamics that incorporates these factors, and a systematic analysis, including stability and sensitivity analyses of the model under different conditions.

Further analyses reveal that adding age-structure to a basic model for malaria transmission in a community does not alter the qualitative dynamics of the basic model, with respect to the existence and asymptotic stability of the associated equilibria and the backward bifurcation property of the model.

However, for ethical and economic reasons this is not a feasible model for malaria vaccinology.

The aim of the present paper is to summarise and update current knowledge on malaria in the African highlands and build a detailed conceptual model for malaria risk factors.

Using a mathematical model for malaria transmission which incorporates the acquisition and loss of both clinical and parasite immunity, we explore the impact of the trade-off between reduction in exposure and decreased development of immunity on the dynamics of disease following a transmission-reducing intervention such as insecticide-treated nets.