Sentence examples for mice was apparently from inspiring English sources

Comparing the size of the evident GJ plaques between WT (Fig. 2A) and Cx30 null (Fig. 2B) mice, the density of Cx26 immunoreactivities in the Cx30 null mice was apparently decreased.

However, the phenotype in the IKO mice was apparently much earlier than in Rab8a/Rab8b double-knockout mice.

The epiphyseal cartilage of the femur in the CSGalNAcT1-null mice was apparently thinner than that of wild-type mice at E18.5.

Thus, on postnatal days 8 13 before the manifestation of ataxia, transmission from PCs to neurons in FN (one of the deep cerebellar nuclei) in the Q129 mice was apparently normal.

The number of lung metastasis in 33D1+ DC-depleted, LPS-treated mice was apparently reduced in comparison with control mice treated with rat IgG or LPS alone visually (Fig.  4a) as well as numerically (Fig.  4b).

Heterozygous Pold1+/− mice were apparently normal and fertile with no detectable developmental abnormalities over 18 months.

Since chronic cigarette smoke exposure did not promote further increases in airspace enlargement in Egr-1−/− mice, these mice were apparently resistant to smoke-induced injury.

The retinal laminar structure and the distribution pattern of GFP-positive cells in SPIG1gfp/gfp mice were apparently normal (data not shown).

IL-6 null mice are apparently normal in terms of their survival, development, skeletal phenotype and response to catabolic PTH [17].

The function of Nanos2 in brain development is unknown, as the Nanos2 knock-out mice were apparently normal except for male infertility [25], and no phenotypic abnormality was reported for Nanos2 transgenic mice, which over-express Nanos2 under the control of Oct4DeltaPE promoter, but the expression was not examined in brain [27].

Given the fact, that the phenotype of Col1a1-Krm2 mise is apparently more severe than the phenotype of Col1a1-Dkk1 transgenic mice reported in the literature [26], it was also important to compare the genome-wide expression analysis of wildtype and Col1a1-Krm2 transgenic osteoblasts to another Gene Chip hybridization experiment, where we have treated wildtype osteoblasts with Dkk1 for 6 hours.