Sentence examples for mice was probably from inspiring English sources

Increased T cell proliferation in vitro or increased expansion of T cells in vivo in HLA-DR3.IFN-γ−/− mice was probably not due to increased expression of HLA-DR3 molecules in HLA-DR3.IFN-γ−/− mice because of comparable numbers of CD4+ T cells were present in both groups of mice in both thymus and spleen (Fig. 7).

As such, the short lifespan of Tsc1c/− /hGFAP2- Cre+ mutant mice was probably a consequence of the onset of status epilepticus, rather than of cachexia and wasting.

The behavioral deficit in the ASIC3 transgenic mice was probably not as a result of an inability to feel the footshock, as footshock sensitivity and vocalization threshold were normal (Fig. 5c,d).

The lack of phenotype in M. sol. in hLPL transgenic mice was probably due to a less pronounced hLPL transgene expression resulting in only slightly elevated FFA and TAG levels (compare Figures 3c and d with Figures 3a and b).

27 Together with other orexigenic and anorexigenic genes, expression of PRCP and MC4R was unchanged in the B1B2KO mouse hypothalamus after treatment with the control diet or the HFD, indicating that the reduced food consumption observed in these mice was probably related to an alternative metabolic pathway not examined in this study.

Since insulin is the initiating autoantigen in the NOD mouse, but other autoantigens such as GAD65 are recognised as the disease progresses [ 24], protection after B 9-23/IFA immunisation in older NOD mice was probably also mediated by bystander suppression mechanisms [ 25– 28].

A practical problem is that the writers will not be able to have their exterminator treat the common areas of the building, like the basement, where the mice are probably coming from, Mr. Kaminsky said.

Thus, the attenuation of DAMGO-induced antinociception in streptozotocin-induced diabetic mice is probably caused by dysfunction in cellular pathways after the activation of G-proteins.

Impairment of adult neurogenesis by AICD in FeCγ25 mice is probably not mediated by cell-autonomous mechanisms because AICD expression is driven by the CaMKIIα promoter, which is active only in mature neurons [30].

This very restricted spatial and temporal pattern of expression of this line of Tet-ΔGR/EGFP transgenic mice is probably due to the site of integration of the transgene within the genome.

The slower mobilization of glycogen stores in transgenic compared to control mice is probably due to plasma insulin, which remained high during fasting, and to the preference for lipid oxidation over glucose oxidation.