Sentence examples for mice identified a from inspiring English sources

Utilizing DCs derived from complement receptor 3-deficient miCR3−/R3−/− mice) identified a requirement for CR3 in the internalization of both sham- and serum-coated particles.

A recent study in mice identified a disease-susceptibility polymorphism of the CTLA-4 gene affecting CTLA-4 splicing in exon 2.

Histological analysis of Rab18 −/− mice identified a delay in closure of the lens vesicle at E12.5 and abnormalities in denucleation of fibre cells in neonates (Fig. 2).

Analysis of the non-tumourous endometrium of tumour-bearing mice identified a series of putative tumour precursor lesions in the surface epithelium of the lumen and in the epithelium of endometrial glands.

This study on prednisolone-induced effects in livers of WT and GRdim mice identified a number of interesting candidate genes and pathways regulated by GR dimers and sheds new light onto the complex transcriptional regulation of liver function by GCs.

A cDNA sequence comparison among susceptible and resistant strains of mice identified a single nucleotide substitution that causes a premature stop codon in the Oas1b transcripts of susceptible mice [ 12, 13].

Slice recordings from PAM mice identified an increase in GABAergic tone (Gaier ED, Rodriguiz RM, Ma XM, Sivaramakrishnan S, Bousquet-Moore D, Wetsel WC, Eipper BA, Mains RE. J Neurosci 30: 13656-13669, 2010).

Defects in IFN signaling can augment tumorigenesis in the 3-MCA-sarcoma model; evidence from USP18 null mice identified an amplified IFN response as compared to wild-type mice, which is thought to provide a less permissive environment for tumors [ 45].

A study in mice identifies a brain adaptation that underlies the compulsive behaviour associated with drug addiction, and which might explain why some drug users behave compulsively whereas others do not.

Indeed, remarkably, genetic deletion of Parp1 rescued normal cerebellar ADP-ribose levels and reduced the loss of cerebellar neurons and ataxia in Xrcc1-defective mice, identifying a molecular mechanism by which endogenous single-strand breaks trigger neuropathology.

Changes in Bmp expression have previously been reported in the limb buds of Lrp4 mutant mice identifying a possible role of Lrp4 in the control of Bmp signalling [15].