Sentence examples for mice manifested a from inspiring English sources

Mut Tg(INS-Alb-Mut) mice, although completely rescued from neonatal lethality that was displayed by Mut mice, manifested a decreased glomerular filtration rate (GFR), chronic tubulointerstitial nephritis and ultrastructural changes in the proximal tubule mitochondria associated with aberrant tubular function, as demonstrated by single-nephron GFR studies.

However, these mice manifested a liver phenotype typical of GSD-1a, characterized by hepatomegaly.

LS- G6pc−/− mice manifested a milder pathological situation in comparison with G6pc−/− mice.

By contrast, Cx3cr1 −/− mice manifested a 4.8-fold reduction in iNOS expression as compared to WT mice (Fig. 4D).

Wild-type mice manifested a continuously increased clinical arthritis score beginning at day 31 until day 41, the last day observed, after primary immunization).

Indeed, WT mice manifested a 1.5-fold higher MyD88 expression in the UUO-induced kidneys as compared with that of Chop −/− mice.

Thus, it appears that CHL1−/− mice manifested an ongoing hyperactive sympathetic nervous system.

Both WT and Cx3cr1 −/− mice manifested an increase in ALT and AST activity 8 weeks after cercariae infection.

Following LI, diabetic mice manifested an altered SP gradient between BM, peripheral blood and limb muscles, accompanied by a depressed recruitment of NK1R-HSPCs to the ischaemic site.

Interestingly, Cx3cr1 −/− mice manifested an opposite phenotype, in which significantly higher levels of Arg-1, a typical marker for Th2 responses, were detected, whereas iNOS expression was significantly suppressed as compared to WT control mice.

Importantly, Chop −/− mice manifested an 80% reduction for LDH activity as compared with that of WT mice, suggesting that Chop deficiency protected tubular cells undergoing UUO-induced apoptosis, thereby prevented the occurrence of secondary necrosis.