Sentence examples for mutant worms show from inspiring English sources

D tyra-2 mutant worms show a chemotaxis index comparable to that of wild-type worms.

tyra-2 mutant worms show defects in the suppression of feeding by 1-octanol, whereas overexpression of TYRA-2 under its own promoter or the zig-3 promoter, which is specifically expressed in the AIM interneurons, can rescue the feeding suppression defect.

In addition, magi-1 mutant worms show defects in memory consolidation, which requires the wild-type MAGI-1 protein in a distinct set of interneurons.

ndk-1 mutant worms show severe reduction of activated, diphosphorylated MAPK in somatic tissues, indicative of compromised Ras/MAPK signaling.

Nevertheless, the mutant worms show nearly normal movement in several assays, though a deficiency in the amplitude of body bending was discernible by video-microscopy [ 29].

We observed that the tyra-2 mutant worms showed a 1-octanol chemotaxis index similar to that of wild-type worms (Fig. 1D), so the difference might result from a cause other than altered 1-octanol avoidance behavior.

For example, the mutant worms showed no apparent motility defects [10], whereas RNAi animals were severely uncoordinated.

lst-4 tm2423) lst-4 tm2423 showed a strong accumutanton of non-acidified, AO-negative cell corpses in the hermaphrodite germline, a similar phenotype observed in dyn-1(ky51) mutants [10] (Fig. 1 A–D', S1), suggesting a defect in cell corpse clearance upstream of phagosome acidification.

ptl-1(ok621) ptl-1 ok621 ptl-1 ok621 worms showed decreandd ptl-1 tm543 ptl-1 tm543 to H2O2 (Fig 1A).

The smc-5 (ok2421 ) mutant worms showed CHK-1 activation similar to wild-type in response to UV treatment, suggesting that DNA damage checkpoint activation is normal in smc-5 (ok2421 ) mutant worms.

In agreement with the reported link between tubulin acetylation and the function of touch receptor neurons in C. elegans, pnk-1 mutant worms showed a decreased touch response, which was also rescued by pantethine feeding (Fig 3B).