Sentence examples for mouse we identified from inspiring English sources

Using the available experimental full-length cDNA data sets for human and mouse, we identified 78 lncRNAs that are either syntenically conserved between human and mouse, or that originate from the same protein-coding genes.

In a screen of potential guidance cues that are expressed at the midline along the rostral course of mDN axons and temporally coincide with their passage (at E12.5 E15.5 in the mouse), we identified Shh as a candidate.

In our studies of CPMV trafficking in mouse, we identified a 54 kD cell-surface CPMV binding protein that was expressed on cells with APC activity (MC57) as well as vascular endothelial cells.

In a retroviral insertion mutagenesis screen in the mouse, we identified GPR110 as an oncogene.

In mouse, we identified that defective sonic hedgehog (Shh) signaling results in early dorsal-ventral epithelial abnormalities prior to the reported defects in septation.

In humans and mouse we identified two TSSs for MECP2, less than 100 bases upstream of the RefSeq annotated start sites of which p1@MECP2/Mecp2 was expressed predominantly in most tissues and p2@MECP2/Mecp2 displayed a stable low level expression in all tissues (expression less than 10 TPM) (Additional file 2: Table S2, Figures  1c, 1d, 2b and 2e).

In addition, using sub-arcsecond radio image of the Mouse, we identify modulations in the brightness distribution of the Mouse that may be associated with the unshocked pulsar wind behind the pulsar.

In mouse, we identify six Dux loci: Gm4981, Dux and LOC100504180 at chromosome 10 B4, and Duxbl and its two tandem copies (Gm10394 and Gm10391) at chromosome 14 A3 [ 16].

Using N-ethyl-N-nitrosourea mutagenesis in mice, we identified a mutant with increased resistance to viral infections because of the presence of hyperresponsive NK cells.

In mice, we identified an essential role for histone 3 lysine 9 (H3K9) dimethylation and the lysine dimethyltransferase G9a in cocaine-induced structural and behavioral plasticity.

Using chromatin interaction sequencing, massively parallel reporter assays (MPRA), and transgenic mice, we identified disease-linked, biallelic HAR mutations in active enhancers for CUX1, PTBP2, GPC4, CDKL5, and other genes implicated in neural function, ASD, or both.