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IDC13 and IDC78 but not IDC16 increased life expectancy of mouse we tested, whereas SR protein depletion is detrimental for survival [24].
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Prior to making transgenic mice, we tested whether ES clones overexpressing Hdac6 show resistance to adenovirus infection.
As 6-OHDA is widely used to induce parkinsonal phenotypes in mice, we tested the functional implication of parkin for PD after 6-OHDA treatments.
Additionally, the mice we tested showed alterations in the ERGs (Figure 4) that were consistent with previous findings [22], [30].
Using mice, we tested the hypothesis that dietary experience during childhood and adolescence affects adult obesity risk.
With limited access to Kv1.1 null mice we tested for confirmation of some of the findings in mceph/mceph.
To evaluate the role of Nrf2 in the hyper-inflammatory phenotype of p47phox−/− mice, we tested whether CDDO-Im would attenuate zymosan-induced lung inflammation in these mice.
In order to address the possibility that these results were limited to DOR-eGFP mice, we tested for acute behavioral desensitization in commercial C57BL/6J mice (Figure 2D).
To investigate anxiety related measures in mGluR5 heterozygous and knockout mice, we tested them in elevated plus maze (EPM) and the light-dark exploration test.
Thus, after confirming that intraperitoneal infection with our B956 strain of WNV induces 100% mortality in B cell-deficient mice, we tested the treatment with si6 and si11.
To test if BK channel inactivation was related to the ataxia observed in BK−/− mice, we tested motor coordination in WT mice by the classical runway test before and after microinjection of paxilline into the vermis.
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CEO of Professional Science Editing for Scientists @ prosciediting.com