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For the mouse model, there was a small increase in 〈z〉 with increasing iodine concentration.

Upon application of the biomaterial in a traumatic or non-traumatic setting in a mouse model, there is a cascade of immune cells that respond to the damaged tissue and biomaterial.

In the mouse model, there is likely some loss of the sugar due to voiding or possibly degradation in the bladder.

As in the mouse model, there was a marked decrease in cysts in the treated rats while trophic form numbers were similar to those from untreated rats.

However, also in our mouse model there is indication that in vaccinated mice sub-clinical infection with influenza virus A/HK/2/68 took place, since weak, short-lived virus-specific CTL responses were observed, which did not protect against challenge infection with the A/H5N1 strain.

Importantly, we found in this study that in the Cngb3−/− mouse model there is some plasticity.

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While spherical treadmills are widely used in mouse models, there are only a few experimental setups suitable for adult rats, and none of them include head-fixation.

Therefore it could be argued that in the hepatic GH resistant mouse models there may be a disproportionate influx of lipids relative to hepatic mass.

All P. falciparum strains tested so far have been supported, indicating that, in contrast to Aotus, Saimiri and previous mouse models, there is no requirement for preliminary adaptation of the parasite to this new host [11], [14], [23], [24], [25].

Also in the studies addressing deletion and inhibition of PERK in neurodegenerative mouse models, there was no clear evidence of canonical UPR activation [ 60, 64, 65].

In contrast to AGPAT2, LPIN1 mutations have been found to be associated with lipodystrophy only in mouse models; there are no known human LPIN1 mutations causing lipodystrophy.

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