Sentence examples for mouse model seems from inspiring English sources

However, recent publications made it clear that the type of mouse model seems to play a crucial role as they differ in extent and type of Aβ pathology [39,35].

The preference for the rat compared to the mouse model seems mainly to be due to the rat's greater size which makes the operational procedures less difficult [ 5].

Therefore, the G3 mTerc−/− mouse model seems more suitable than wild‐type aging mice to study the effects of telomere shortening on microglia.

Although many different animal models for FAS have been developed [ 90], the mouse model seems to correlate best to the effects of prenatal alcohol exposure observed in humans [ 91].

Using semantic similarity to identify diseases that resemble mouse models seems reasonable; using this to predict disease genes is most definitely GBA and suffers all the same potential pitfalls (and then some).

Perhaps the limitation of current mouse models seems to reside in the lack of models that show a flare-and-wane-type of chronic disease that is most common in human SLE: all mouse models represent severe cases with increased severity of pathology over time.

They chose inflammation as a field of medical research, in which human data are available and in which the mouse models seem to have a very good mechanistic resemblance to the human disease situation.

Phenotypically, most of the existing mouse models seem to be a mixture of OC and GS, including mice deficient in Scrib (Carnaghan et al., 2013; Murdoch et al., 2003), Aebp1 (Layne et al., 2001; Danzer et al., 2010), Bmp1 (Suzuki et al., 1996), and Tfap2a (Brewer and Williams, 2004b) (description summarized in Table 3).

The MUC1 TG mouse tumor model seems to back up the idea of tolerance against MUC1 in humans, but the modifications of MUC1 glycosylation observed in human cancer cells were not taken into account in these studies.

To investigate the causes of early metastatic relapse after surgery, the team created a mouse model that seems to mirror the immunological detente keeping in check dormant, disseminated tumor cells in breast cancer patients.

At first sight the model seems to be in conflict with experiments showing that single UCNEs can drive tissue-specific gene expression in mouse or zebrafish embryos.