Sentence examples for mouse embryo we from inspiring English sources

To perform systematic clonal analysis in the developing mouse embryo, we applied a method that relies on the use of ubiquitously expressed knock-in alleles and is suitable for non-invasive permanent cell labelling during embryonic development [10].

Although there have been few reports about expression in the developing mouse embryo, we found our results to be generally consistent with published CA data.

To determine whether this might be so in the mouse embryo, we followed the segregation of Cdx2 transcripts during divisions that generate cells of distinct fates.

Using the same half-embryo inhibitor assay in the mouse embryo, we found that exposure to 100 μM XAV939 delayed the pace of mLfng oscillations as compared to control DMSO-treated E10.5 half PSM explants.

Because these subdivisions are not defined as sharply as before and distinct neuronal populations have already arisen or are arising in the mid-/hindbrain region of the E12.5 mouse embryo, we additionally defined individual neuronal populations and new subdivisions of the regions (compartments) tel-, di-, mesencephalon, MHB, met- and myelencephalon and also spinal cord.

If cardiac differentiation in EBs is equivalent to cardiac differentiation in the mouse embryo, we would expect that an early depletion of Islet-1expressing cells would lead to a general depletion of SHF derivatives and this should impact the ratio of atrial to ventricular cell types.

And consistent with previous findings in human cells and mouse embryos, we found proximal-site deamination using gRNA-2 (Fig. 1C) (Liang et al., 2017).

In mutant Hes1 mouse embryos, we found that the thyroid gland was hypoplastic and hypofunctional.

In previous analyses of histone methylation dynamics in mid-gestation mouse embryos, we documented marked changes in H4K20 methylation during cell differentiation.

As a first step towards assaying circadian regulatory cycles (CRCs) in mouse embryos we surveyed the expression of CRGs during post-implantation development.

In order to identify when and where the c-fos intronic promoter is active in mouse embryos, we constructed the fiZ transgene (fos intron LacZ, fig. 4A, see methods).