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Calculations for small many-electron model systems indicate the existence of finite two-body parameters that produce the numerically exact wave functions for ground and excited states.

Ab-initio calculations in CNT model systems indicate that the Diels-Alder addition of butadiene is a feasible process and that subsequent oxidation reactions may result in the formation of the anhydride moiety.

Studies in model systems indicate that neural activity may also promote dependency on internal initiation of translation.

However, cellular model systems indicate arsenic can induce malignant transformation of human prostate epithelial cells in vitro.

Studies in breast and prostate carcinoma model systems indicate that tumor cells from metastasis suppressed kai1 or kiss transfectants, as well as their and metastatically competent control transfectants, arrived in the distant organ at comparable frequencies and with equivalent viabilities.

Studies in model systems indicate that this resistance may often depend on the acquisition of enhanced cross-talk between ER and growth-factor pathways that allows the disease to circumvent the need for steroid hormones [ 1].

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Comparison of the two model systems indicates that only a dedicated Cu ZrOxHy interface with in situ formed and reversibly hydroxylated sites (accessible only from initially (inter)metallic Cu/Zr species at the surface) leads to water activation, total oxidation of intermediate formaldehyde, and enhanced CO2 selectivity.

On the other hand, cells with very high (>200 DMs) were extremely rare in our model systems, indicating some degree of selection against cells with an extremely high DM copy-number.

Support for this model includes the multiphoton laser experiments shown here (Figures 11 and 12), as well as evidence in other model systems indicating that loss of UNG2 results in increased sensitivity to ionizing radiation [23], [58], which primarily produces double-strand breaks.

Both wingless and Ras activated form transgenes were lethal when expressed in larvae, and reduced life span when expressed in adults, consistent with results from other model systems indicating that the wingless and Ras pathways can promote senescence.

Concordant with a proposed non-proteolytic function for PSA, there is evidence from tauopathy model systems indicating that proteolytic degradation is not the mechanism by which PSA reduces protein deposition and rescues toxicity.

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