Sentence examples for mice which suffer from inspiring English sources

This modest phenotype is different from that of lck-Cre/survivinfl/fl mice, which suffer from an almost complete block of DN to DP development [18].

Along those lines, enhanced eryptosis is observed in Klotho deficient mice which suffer from excessive 1,25(OH)2D3 formation [ 218, 219].

This is supported by the phenotype of B- myb knockout mice, which suffer early embryonic death due to proliferation defects of the cells of the inner cell mass.

Analyses were performed in human tumor necrosis factor transgenic (hTNFtg) mice, which suffer from chronic destructive arthritis, and wild-type mice.

These results were confirmed in apolipoprotein-E and angiopoietin-like 3-deficient mice, which suffer from hypercholesterolemia and hypolipidemia, respectively [ 115].

In this report we used Meis1-GFP transgenic mice to purify mesangial cells from normal mice and from db/ db mice, which suffer diabetic nephropathy.

Similar to the findings in epidermal-specific desmoplakin-deficient mice, which suffered from skin blistering, desmosomes were not anchored to intermediate filaments (Vasioukhin et al. 2000).

Second, limiting the analysis to those mice which suffered severe initial injury with day 2 BUNs greater than 200, we found no difference in the rate or extent of recovery of kidney function between WT and Clic4 null mice.

After Treg depletion, organ-specific autoimmune diseases, especially autoimmune gastritis, predominantly developed in +/+, at a lesser incidence in skg/+, but not in skg/ skg BALB/c mice, which suffered from other autoimmune diseases, especially autoimmune arthritis.

We crossed mice expressing human Aβ to mice lacking or overexpressing PrPC or a soluble variant thereof to evaluate if the impact of PrP is persistent also in another AD mouse model which suffer from Aβ-dependent learning and memory deficits (Serneels et al, 2009).

The effects of SOD1 loss of function on motor neurons have been recently readdressed through the study of Sod1−/− mice (20, 21), which suffer from a severe progressive denervation of hindlimb muscles, leading to striking motor phenotypes (20).