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Sick mice were distinguished from healthy ones by enlarged abdomen at age of 10 days.

ADAM17-null and control mice were distinguished by PCR and functional screening.

Genotyping for Cre recombinase was performed using the following primers: forward 5'-CGATGCAACGAGTGATGAGG-3' and reverse 5'-GCATTGCTGTCACTTGGTC-3'. Cre-expressing mice were distinguished by the presence of a 288bp amplicon from ear punch DNA samples.

Mitochondria-rich cells that expressed pendrin in Slc26a4+/− mice were distinguished from ribosome-rich cells in Slc26a4+/− and Slc26a4−/− mice by their dense actin expression near the apical membrane (Fig. 7G).

Histopathological examination indicated that neoplastic lesions from all groups were adenomas.Yet, lesions from HCD-post-tumor-onset Kras G12D mice were distinguished by the presence of nests of cells containing large atypical nuclei, indicating a more advanced tumor grade.

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As discussed previously, immunized and unimmunized mice are distinguished with 100% efficiency.

How these uni-strand clusters in D. melanogaster and piRNA clusters in mice are distinguished from very similar looking canonical mRNA-producing loci and how those RNAs are funneled into the piRNA processing machinery still remains to be answered.

Fatty acid metabolism in white adipose tissue from PPARγ agonist treated ob/ ob mice was distinguished by increased concentration of long carbon chain fatty acids and an increase in the monounsaturated fatty acid products of the Δ-9 desaturase.

The body and footprints of the mouse are distinguished from the background and from each other based on their color or pixel intensity.

In direct co-cultures, mouse vs. human cells were distinguished by immunodetection with human-specific/hESC-specific and mouse-specific antibodies (Supplementary Fig. S4 and see below).

hESCs were distinguished from mouse cells by using a species-specific antibody to the cell-surface marker M-cadherin for murine and the nuclear marker NuMA for human cells.

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