Sentence examples for mice were designed from inspiring English sources

The HA0 cleavage loop peptides used for intranasal immunization of mice were designed and synthesized from specific conserved regions of influenza A/H1N1 & A/H3N2 viruses.

To investigate the role of nuclear CaMKII in cardiac function, transgenic mice were designed and generated to target the expression of a CaMKII inhibitory peptide, AIP (KKALRRQEAVDAL), to the nucleus.

To validate the pathophysiological consequences of the interaction between BRAFV600E and MST1 in vivo, animal experiments using thyroid-specific transgenic BRAFV600E mice (Tg-BRAFV600E mice) and MST1 null mice were designed.

Human IL-32α Tg mice were designed to overexpress human IL-32α by using CAG promoter.

Our studies using the DO mice were designed to examine nutrigenetic or gene × diet interactions and the overall design is outlined in Figure 1.

These transgenic mice were designed to express cis-acting genomic regulatory elements in a manner that faithfully recapitulates endogenous PER2 expression.

In this study, a combined treatment of Aβ1-42-bone marrow derived dendritic cells (BMDCs) with intraperitoneal injection of splenocytes from young mice was designed as a novel immunotherapy for AD in APPswe/PSEN1de9 transgenic mice models.

To test this hypothesis, a novel cue-potentiated feeding protocol adapted for use in mice was designed and validated, and then the effects of pharmacologic ghrelin receptor (GHSR) antagonism and GHSR transcriptional blockade (as occurs in GHSR-null mice) were assessed.

The running protocol applied to the mice was designed to provide a strong, but non-exhaustive metabolic stimulus, resembling intensive endurance exercise.

The SCID-hu mouse model, in which human lymphoid organs are implanted into severe-combined immunodeficient (SCID) mice, was designed by McCune et al. [1] to provide a small animal model for the study of human hematopoiesis.

Thus, all cells of Z/AP and Z/EG mice are designed to exhibit a binary readout of Cre activity, expressing β-galactosidase by default and activating expression of hPLAP or EGFP respectively upon exposure to Cre.