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We studied the effects of chronic administration of Tβ4 on the skeletal and cardiac muscle of dystrophin deficient mdx mice, the mouse model of Duchenne muscular dystrophy.
However, myosphere cells inoculated into the damaged brain of Experimental Autoimmune Encephalitis (EAE) affected mice, the mouse model of multiple sclerosis, did spread in the brain and a fraction of them gained neuronal morphology and expressed neuronal markers [28].
Haploinsufficiency for tumour development was reported for the Nbn knockout mice, the mouse homologue of NBS1.
As they generated more oncomice and knockout mice, the mouse models with individual gene target provided even more information when crossed with mice with various genetic backgrounds.
Early studies have shown elevated NF κB levels in skeletal muscle of mdx mice, the mouse model for Duchenne muscular dystrophy (DMD), and in inflammatory myopathies.
We selected non-obese diabetes (NOD) mouse, the most popular and extensively studied mouse model of SS [ 13, 14] and interleukin-14 alpha-transgenic (IL-14α-TG) mice, the mouse model for primary SS [ 15, 16].
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Inter-generationally, there are more mice, the mice are more hungry, and there is less cheese in life's maze.
And when the insects were allowed to feast on mice, the mice developed antibodies against SP15.
When researchers inhibited ENaCs in mice, the mice couldn't taste salt.
In comparison with the obese control mice, the mice treated with S. alata showed a significant reduction of liver glucose-6-phosphatase (G6Pase) and phosphoenolpyruvate carboxykinase (PEPCK) proteins.
When the researchers removed the VNO from normal female mice, the mice acted just like the TRPC2 mutants.
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