The markers with allele frequency significantly different between the high fat content and the low fat content sheep (62 markers) were different from the markers discriminating the sheep on the basis of milk protein percentage (207 markers).
For example, at the p<0.05 level there were 151 variables where either cell count or the intensity of cell surface markers were different between children with autism and controls (Table 2).
As expected, a number of markers were different between those with and without impaired renal function in the control group (Table 3).
20 Wilcoxon Mann–Whitney tests were used to test whether particular markers were different between Japanese and Caucasian GCs, using tumour content-adjusted median immunohistochemical (IHC) values (significance p<0.05).
This distribution was similar to the percentages observed for all markers of the three types, but the distributions of the three types of segregation distortion markers were different for each LG.
To determine whether the associations between p53 mutation and the various clinical markers were different in p53β- or p53γ-positive tumours, data were stratified by p53β and p53γ expression status.
