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Fifteen subjects had a congenital visual deficiency, 7 with total blindness (CBS: completely blind subjects) and 8 with some residual vision (BRV: blind with residual vision) and 12 had normal vision (see Tables 1 and 2).
Mutations in the gene encoding the protein RPE65 are associated with several forms of inherited retinal dystrophies, such as autosomal recessive retinitis pigmentosa (RP) [1] and autosomal recessive childhood-onset severe retinal dystrophy [2] which are characterized by profound visual deficiency, night blindness and reduced or non detectable electroretinogram (ERG).
In one patient with orbital RMS visual deficiency present prior to RT declined leading to amaurosis.
Visual deficiency results in a significant economic and social disadvantage in affected individuals, their families, and society in general.
Patients with a visual deficiency have more frequent medical visits, and many need assistance to perform daily life activities.
Additionally, many coexistent neurological alterations are present in scoliosis patients, such as visual deficiency [ 6] and decreased postural stability [ 7, 8].
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The paper includes review and discussion of electrode site location, methods of normalization, data reliability, and factors that can affect sEMG signals from this region, including noise, physiologic artifact, stress, visual deficiencies, and pain.
Using linear programming, they figured out an algorithm that identifies which filters provide the most optimal solutions to visual deficiencies.
Visual deficiencies can be compensated for with optical aids.
Cav1.4 knock-out mice support this view as these mice show severe visual deficiencies [13,14].
Individuals with poor natural visual acuity or an age ≥40 years can reliably compensate their visual deficiencies by using optical aids.
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CEO of Professional Science Editing for Scientists @ prosciediting.com