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Objectives: The present study aimed at evaluating myocardial function at the early phase of TBI using conventional 2D echocardiography and complementary STE analysis.

These techniques offer assessment of myocardial function at presentation together with monitoring of haemodynamic response to fluid expansion.

Although resting echocardiography provides information about myocardial function at a static state, the dynamic response of the myocardium to stress can only be revealed by stress testing [ 6].

Here, we review emerging evidence that suggests testosterone influences myocardial function at the cellular level by modifying processes involved in intracellular Ca2+ homeostasis.

These findings demonstrate that GDX influences critical mechanisms involved in Ca2+ homeostasis and suggest that testosterone modulates myocardial function, at least in part, by effects on individual ventricular myocytes in rodent models.

Especially in children, generally paced from a very early age and typically having normal myocardial function at the initiation of pacing, as well as having a perspective of life-long pacing, the preservation of cardiac function during chronic ventricular pacing should take high priority.

Similar(54)

Animals treated with argon presented also a significantly better recovery of systolic myocardial function, as represented by the higher SF at 96 hr compared to controls (Table 87).

Based on the relationship between myocardial perfusion and myocardial function as previously described in patients with idiopathic dilated cardiomyopathy [ 9– 11], we hypothesized that myocardial perfusion is associated with changes in regional myocardial function after MI.

Myocardial function was measured at multiple, independent end points.

Myocardial function was assessed at vasopressin concentrations of 0, 10, 25, 50, 100, 200, 400, and 500 pg/ml.

Echocardiographic comparison of baseline myocardial function with that at 1 month and 1 year using r-ANOVA is shown in table 2. There was gradual and progressive deterioration in all myocardial functional parameters over time in children who received anthracycline.

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