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Myocardial performance at the cellular level is associated with the number or functional capacity of mitochondria.
Myocardial performance at peak stress was improved and maintained to 30 minutes, and postischaemic stunning was abolished with sitagliptin [ 83].
Our results indicate that patients with mild PHPT without any other known cardiovascular risk factors have a higher systolic myocardial performance at baseline, which seems to be associated with the level of PTH and Ca++.
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The results of hemodynamic measurements in this study indicated that diastolic and systolic myocardial performance was depressed at the eighth week of diabetes, as determined by significant reductions in ± dP/dtmax.
Even in the absence of CAD, patients with diabetes show changes in myocardial performance that put them at risk for heart failure (diabetic cardiomyopathy).
However, after GLP-1 infusion during hyperglycemia, myocardial performance was enhanced both at peak stress (11.9 ± 2.7 [GLP-1 HHC] versus 11.0 ± 2.9 cm/s [control HHC], p = 0.01) and at 30 min after dobutamine stress (6.7 ± 0.6 [GLP-1 HHC] versus 6.1 ± 0.7 cm/s [control HHC], p = 0.02) (Fig. 5b).
Levosimendan improves myocardial performance by increasing myofilament calcium sensitivity, without raising intracellular calcium and AMP concentrations.
The transitional circulation and its effect on myocardial performance are poorly understood in preterm infants.
In the post-myocardial infarction (MI) patient with coronary artery disease (CAD) and left ventricular dysfunction (LVD), ischemia and adverse remodeling hinder myocardial performance and increase electrical instability.
Further study is necessary in order to explore the effect on myocardial performance independently from vascular function.
Also observed was a negative correlation was with tissue doppler myocardial performance index (left) (N = 74; r = -0.226; p = 0.05).
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com