Sentence examples for mutants were positive from inspiring English sources

Whereas wild-type and genetic capsule synthesis mutants were positive and negative for agglutination, respectively, C7 treated UTI89 did not agglutinate, and whole cell sonicates had only weak reactivity (Table 3).

Indeed, endothelial and endocardial cells in bar mutants were positive for TUNEL staining and show typical morphological features of apoptotic cells at this stage.

All single mutants were positive for calcofluor staining and colony morphologies with Congo red staining showed that all stained to a higher degree than the SM ancestor, but there were differences.

Interestingly, in contrast to the floating T-bar super-aggregates in srpk79D mutants (Johnson et al., 2009; Nieratschker et al., 2009), these axonal BRP spots in aplip1 mutants were positive for Syd-1 (compare Figures 1C, 7B).

PZase assay showed that the 5 mutants were positive for the enzyme activity, which is consistent with the above pncA sequencing results and also ruled out a pncA promoter or regulatory mutation that could result in lack of PZase enzyme activity as a possible cause of the PZA resistance in the 5 mutants.

The ubiquitinated protein aggregates in bchs mutants are positive for Ref(2 P [ 244].

We found that cardiovascular tissues from bar mutants are positive for 8-hydroxy-2′-deoxyguanosine (8-OHdG), a free radical-induced oxidative lesion in DNA.

This result was consistent with the induced or enhanced expressions of AS, IGPS and TS genes in the tryptophan and serotonin biosynthetic pathway, and the lesion mimic phenotype of spl5 mutant was positive correlated with the content of serotonin.

By contrast, the cheR1 mutant and complemented mutant strain were positive in swarming (Figure 3C and D) and twitching (data not shown) in both, the PA14 and PA01 strain background.

The genomic DNA samples from the 5 PZA-resistant mutants that were positive for PZase and did not have pncA mutations were subjected to whole genome sequencing using Illumina HiSeq 2000 machine.

Applying a digital PCR assay, Oshiro et al. detected that serum samples from 23%% of PIK3CA-mutant patients were positive for this mutation [ 33].